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논문 기본 정보
- 자료유형
- 학술저널
- 저자정보
- 발행연도
- 2022.12
- 수록면
- 44 - 68 (25page)
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초록· 키워드
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T-helper-17 (Th17) cells and related IL-17-producing (type17) lymphocytes are abundant at the epithelial barrier. In response to bacterial and fungal infection, the signature cytokines IL-17A/F and IL-22 mediate the antimicrobial immune response and contribute to wound healing of injured tissues. Despite their protective function, type17 lymphocytes are also responsible for various chronic inflammatory disorders, including inflammatory bowel disease (IBD) and colitis associated cancer (CAC). A deeper understanding of type17 regulatory mechanisms could ultimately lead to the discovery of therapeutic strategies for the treatment of chronic inflammatory disorders and the prevention of cancer. In this review, we discuss the current understanding of the development and function of type17 immune cells at the intestinal barrier, focusing on the impact of microbiota-immune interactions on intestinal barrier homeostasis and disease etiology.
목차
ABSTRACT
INTRODUCTION
MICROBIOME-MEDIATED TYPE17 IMMUNE RESPONSE
MICROBIAL AG-SPECIFIC PRIMING OF TYPE17 IMMUNITY
REGULATION OF TYPE17 IMMUNITY BY NON-ANTIGENIC MICROBIOTA-DERIVED MOLECULES
DYSREGULATION OF HOST-MICROBIOTA INTERACTIONS
CONCLUSION
REFERENCES
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